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NOMENCLATURA |
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- 1901 -
Karolyi , “traumatic neuralgia”
- 1907 -
Marie y Ptietkieviez , “bruxomania”
- 1931 -
Frohman , “bruxism”
- 1962 -
Drum , “parafunction”
- 1971
- Ramfjord y Ash , “centric and eccentric bruxism”.
- Oclusión
2ª Ed. Saunders. Philadelphia, 1971.
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DEFINITION |
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The
habit of gritting or rubbing of teeth, at day or night-time, with
different intensity and persistence levels, in a certain space of
time, unconsciously away from functional movements (parafunction).
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Neurophysiological
disorder of the jaw movements which, in a progressive manner, destroys
the dental tissue.
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The
clinical repercussions can go far beyond the wearing out of teeth,
affecting dental support structures, the cervico-cranial muscles and
TMJ structures.
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Affects
both male and female, teenagers and grown ups (although the latter
may have begun in their teens), even very young children (do not mix
this up with physiological attrition in deciduous dental pieces).
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ETIOLOGY |
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The
origin of bruxism has not been fully clear or explained.
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It
seems that it obeys to multiple etiopathogenical factors.
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Among
them, there are two which outstand above the others:
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Occlusal
interferences
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Mental
factor
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The
most pathogenical interferences are:
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Psychical
or mental factors work as potentiators of the symptoms (contributing
factors). They are:
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Anxiety
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Stress
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Stressful
situations can potentiate this habit in a specific manner
- Relaxing
situations can reduce such habit in an occasional manner
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DIFFERENTIAL DIAGNOSIS |
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ABRASION:
The lost of dental substance due to gritting or rubbing. (Bruxism
and energetic brushing of teeth and gum)
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ATTRITION:
The lost of dental substance due to functional wearing out or erosion
(chewing).
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EROSION:
The lost of dental substance because of chemical substances (vinegar,
lemon, acid regurgitation of the stomach).
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CLASSIFICATION |
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There
are two types of bruxism:
- CENTRIC
- Squeezers
or tighteners
- Especially
at night-time
- Eroding
area limited to occlusal face
- Lesser
dental erosion
- Major
muscle affectation
- ECCENTRIC
- Rubbing
devices
- Night-time
- Eroding
area exceeds occlusal face
- Major
dental erosion
- Lesser
muscle affectation
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CLINIC |
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- Dental
wearing out or erosion:
- In
centric bruxism: inverted cusps and wearing out of the neck of the
teeth.
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- In eccentric
bruxism: the wearing out process is away from the functional area.
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In
a study done by Powell and Zender in 1965 dental contacts were registered
during sleeping hours.
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The
non-bruxists standard registers presented an average 260 dental
contacts in 8 sleeping hours. In the bruxists registers there were
some 150 to 1500 dental contacts per hour (1200 to 12000 in 8 sleeping
hours).
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- Occlusal
trauma
- In
centric bruxism
- It
can cause periodontitis and dental mobility.
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- Dental
hypersensitivity
- In
centric bruxism due to the loss of enamel in the neck of dental
pieces.
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- Pulpitis
and pulpals necrosis
- When
the pulpal retracting mechanism fails (compensation mechanism before
the erosion)
- In
eccentric bruxism
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- Bone affectation
- Centric
bruxism: Bone reabsorption.
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- Eccentric
bruxism: Bone condensation and exostosis.
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- Muscular
affectation
- Especially
in centric bruxism
- Pain
and sensitivity in muscle elevators (maseter, temporal)
- Functional
limitation
- It
can affect the neck muscles
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- ATM affectation
- Due
to loss of muscular synergy (especially from the meniscal instalment
of the external Pterigoid)
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- Loss of
the vertical dimension of occlusion
- In
eccentric bruxism
- Because
of dental erosion
- Esthetic
problems
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DIAGNOSIS |
- Locating
the dental erosions by buccal exploration
- Associated
clinic to parafunctional habit
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TREATMENT |
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- REVERSIBLE
- Contributing
control factors
- Control
of the psychic factors that work as potentiators of the symptom:
- Psichoterapy
- Relaxation
- Yoga
- In
the daytime tightener: deprogram such habit (tongue-palate exercises)
- Medicine
- Anxiolitics:
- Diazepam
(Valium ®)
- Clorazepato
dipotasic (Tranxilium ®)
- Clonazepam
(Rivotril ®)
- Muscle
relaxers:
- Tetrazepam
(Myolastan®)
- Metocarbamol
(Robaxisal ®)
- Occlusal
splints
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Especially
in night-time bruxism
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They
are also called splints or discharging plaques
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Built
of acrylic, generally the upper unit
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They
must fully cover the occlusal surface and the incisal edges
of the incisors and canines
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The
width of the splint should be about 2 mm and its surface ought
to be flat.
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Maximum
bilateral contacts must be obtained between the lower vestibular
cusps and the plaque.
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They
are constructed with canine guide so that in laterality they
only go into contact with the canines.
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The
objective of its use is:
- Ovoiding
dental erosion
- Muscular
decontraction
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- IRREVERSIBLE
- Occlusal
adjustment
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Apply
only at initial stages of bruxism (very difficult to detect).
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It
consists in the detection and elimination of occlusal interferences,
potentially responsible for installing the medical profile
or symptom.
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Getting
rid of the interferences is done through the selective carving
or shearing technique
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The
occlusal adjustment will be, in certain way, a prophylactic
method that should be applied in dental treatments with the
main intention of avoiding the genesis of interferences which
could potentially spark off bruxim.
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Once
the habit is established, Occlusal adjustment is practically
irrelevant.
- Oral
rehabilitation
- In
cases of great loss of vertical dimension or affectation of the
TMJ
- It
must be based on physiological parameters: Hinge axis, RC, condylar
paths, Bennett movement and type of occlusion.
- Its
realization is very complex and does not guarantee getting rid of
bruxism.
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